BugTracker
Active member
I'm looking at some studies on embryonic peptides and the language is kinda throwing me off. Could someone explain a few things?
Here's an example:
"Humanofort (EP) shows an adaptive pattern, reacting to the initial levels and adjusting towards normal ranges. This was consistent across all measures, including insulin and IGF-1, both involved in regulating fat metabolism. Surprisingly, HT's adaptive effect was also seen in cortisol, DHEA, and even some immune markers like PSA and CEA."
"EP might work by interacting with the hypothalamic-pituitary-gonadal/adrenal axes. EP also altered cortisol, steroid hormones (DHEA and Testosterone), and antigens (CEA and PSA)."
So, my understanding is that it might lower fasting insulin, raise IGF-1 when it's low, help with lipids, lower cortisol (and raise DHEA?), and lower PSA and CEA. Am I on the right track??????
I read that it works by:
- normalizing adrenal cortex activity at the cellular level
Thanks!
Here's an example:
"Humanofort (EP) shows an adaptive pattern, reacting to the initial levels and adjusting towards normal ranges. This was consistent across all measures, including insulin and IGF-1, both involved in regulating fat metabolism. Surprisingly, HT's adaptive effect was also seen in cortisol, DHEA, and even some immune markers like PSA and CEA."
"EP might work by interacting with the hypothalamic-pituitary-gonadal/adrenal axes. EP also altered cortisol, steroid hormones (DHEA and Testosterone), and antigens (CEA and PSA)."
So, my understanding is that it might lower fasting insulin, raise IGF-1 when it's low, help with lipids, lower cortisol (and raise DHEA?), and lower PSA and CEA. Am I on the right track??????
I read that it works by:
- normalizing adrenal cortex activity at the cellular level
Thanks!